Bureau Briefs
ACADEMIC SENATE
The faculty of the UCLA College, followed by the faculties of
the Schools of Arts and Architecture and Theater, Film and Television,
will soon be voting on a proposal to institute a diversity requirement
for undergraduates. In the past, other proposals have failed. “This
one is different, and if people will read it, I think they will
be impressed,” Academic Senate chair Kathleen Komar told the
Senate’s Legislative Assembly when it met Nov. 16. “The
issue of a diversity requirement has been carefully considered over
several months by a group of faculty and students who have really
hammered this out,” she said. To get an overview of the diversity
requirement and comments by Raymond Knapp, chair of the General
Education Governance Committee in the College, read the Voice of
the Faculty: http://www.senate.ucla.edu/SenateVoice/Issue8_Nov04/home.htm.
MURPHY HALL
Chancellor Albert Carnesale has appointed Gerald S. Levey, vice
chancellor of medical sciences and dean of the David Geffen School
of Medicine, to the 29-member Independent Citizens Oversight Commission
that will govern the stem cell research institute approved by California
voters Nov. 2. Under Proposition 71, the chancellors of the five
UC campuses with medical centers each appointed an executive officer
of his or her campus to the commission. In addition, Lt. Gov. Cruz
Bustamante appointed UC Berkeley Chancellor Robert J. Birgeneau
as one of the four other California university representatives to
the commission.
GEFFEN SCHOOL OF MEDICINE
UCLA scientists have devised a novel way to repair one of the
genetic mutations that causes ataxia-telangiectasia (A-T), a disorder
that devastates the neurological and immune systems of one in 40,000
children. A-T usually strikes children before age 2 and confines
them to a wheelchair by age 10. Many lose their ability to speak,
and die in childhood. Richard Gatti, professor of pathology and
laboratory medicine, and Chih-Hung Lai, a postdoctoral researcher
at the David Geffen School of Medicine, created a new strategy for
tricking the mutated A-T gene into overlooking certain types of
mutations called premature termination codons (PTCs). “PTCs
are like irregular stop signs located in the middle of the block,”
Gatti said. “They stop traffic before it reaches the intersection.
We made these stop signs invisible, so traffic continues until it
sees the proper stop sign at the end of the corner.”
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